Home  >  Products  >  CLDN5 (claudin 5 (transmembrane protein deleted in velocardiofacial syndrome)) Blocking Peptide (the C terminal of protein) (100ug)
CLDN5 (claudin 5 (transmembrane protein deleted in velocardiofacial syndrome)) Blocking Peptide (the C terminal of protein) (100ug)

CLDN5 (claudin 5 (transmembrane protein deleted in velocardiofacial syndrome)) Blocking Peptide (the C terminal of protein) (100ug)

Supplier: Aviva Systems Biology Incorporated
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This is a synthetic peptide designed for use in combination with anti-CLDN5 antibody (Catalogue #: ARP33627_P050) made by Aviva Systems Biology. It may block above mentioned antibody from binding to its target protein in western blot and/or immunohistochecmistry under proper experimental settings. There is no guarantee for its use in other applications. Please inquire for more details.
Presku: AAP33627
Size: 100 ug
Weight: 23kDa
Gene: 7122
Format: Lyophilized powder
Target: CLDN5 is a member of the claudin family. Claudins are integral membrane proteins and components of tight junction strands. Tight junction strands serve as a physical barrier to prevent solutes and water from passing freely through the paracellular space between epithelial or endothelial cell sheets. Mutations in this gene have been found in patients with velocardiofacial syndrome.This gene encodes a member of the claudin family. Claudins are integral membrane proteins and components of tight junction strands. Tight junction strands serve as a physical barrier to prevent solutes and water from passing freely through the paracellular space between epithelial or endothelial cell sheets. Mutations in this gene have been found in patients with velocardiofacial syndrome. Publication Note: This RefSeq record includes a subset of the publications that are available for this gene. Please see the Entrez Gene record to access additional publications.
Alternative names: AWAL; BEC1; CPETRL1; TMVCF
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